In Romania, the incidence of CRC in is We reviewed a series of studies that are related to colon cancer and studied the epithelial-mesenchymal transition at the front of tumor invasion EMT.
Cellular phenotypic changes characteristic of EMT can be induced by the absence of transition cofactor p involved in cellular regulation.
Loss of syndecan-1 marker is associated with local tumor stage and metastasis. Modulators of protein kinase resistance was associated with changes in genes involved in EMT including vimentin hyperexpression and genes involved rectal cancer incidence invasion N-cadherin with a decrease expression of genes involved in epithelial cell adhesion E-cadherin.
Progression in colon cancer is characterized by activating mutations in Ras genes and tumor growth factor action. Vimentin expression associated with EMT initiates molecular program.
One of the characteristics of EMT is the loss of E-cadherin. TGF-β transforming growth factor beta induces epithelial-mesenchymal transition in colon cancer cell lines with the microsatellite stability, inducing cell invasion and migration.
EMT is a critical early event involved in invasion and metastasis of colorectal cancer, characterized by the presence of markers specific to each phenotype, epithelial or mesenchymal. Multiple biomarkers involved in the induction of EMT may represent future therapeutic target in the treatment of colonic neoplasia.
Glimelius B, Oliveira J.
What is Colorectal Cancer?
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